glutathione peroxidase a selenoenzyme Rational redesign of the active site of selenosubtilisin with strongly enhanced activity Posttranscriptional Defects of Selenoenzymes |

Notably, these findings collectively highlight that NPC ferroptosis is a confirmed promotor of IDD by impairing the viability of NP-residing cells as well as damaging the extracellular matrix (ECM) 7,8 , while the operation-induced physical and biochemical insults could significantly promote the ferroptosis cascades in NPCs to enhance the degenerative phenotype as well as aggravate the IDD symptoms

V odborn literatue je popisovna jeho role v procesech souvisejcch s oxidativn rovnovhou a bunnou ochranou ped oxidanm stresem

Although host metabolic adaptations that are shaped by evolutionary pressure in response to endemic viruses are conserved 7 , they may be dysfunctional in the context of emerging zoonotic infections

Journal of Alzheimers Disease, 42(4), 10791149