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The pathological hallmarks of AD include glucose uptake reduced in the brain, the accumulation of A and neurofibrillary tangles (Patrick, 2019)

96 Furthermore, the use of dual agonists in NASH treatment has been explored, combining GLP-1 with glucose-dependent insulinotropic polypeptide (GIP) agonism (tirzepatide) or GLP-1 with glucagon agonism (cotadutide)