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most oral glutathione is rapidly broken down by intestinal peptidases, necessitating either liposomal formulations, precursor compounds (NAC, milk thistle), or intravenous administration for systemic delivery

SMs secrete various types of proinflammatory cytokines, overproduce reactive oxygen species (ROS), and then harass the arthritic synovial microenvironment via complicated inflammation networks 5,6

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In pDCs, SLC15A4 is required for TLR7- and TLR9-mediated IFN-I productions, as the mutation of SLC15A4 indicates that AP-3, as well as the BLOC-1 and BLOC-2 Hermansky-Pudlak syndrome proteins are essential for pDC signaling through TLR7 and TLR9