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indicate that while inhibiting NETs formation is possible, it does not alleviate the extent of kidney injury (102)

Mice were monitored daily, and clinical scoring was performed as mentioned before with a 6-point scaling system

Data from the simultaneous measurement of extracellular acidification and oxygen consumption revealed that in glucose-fed human iPSC-derived astrocytes glycolysis and oxidative phosphorylation contribute both around 50% to the ATP regeneration and that accelerated glycolysis can compensate for an impairment of mitochondrial ATP regeneration [122]

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