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With RSL3-induced reduction in pNPM1 levels, DNA damage sites (marked by -H2AX foci) were not repaired due to limited availability of pNPM1

The accumulation of lysophosphatidylcholine in hepatocytes can cause apoptosis by activating the death receptor DR5 while promoting macrophage activation through pro-inflammatory exosomes [36]

It stimulates the proliferation and differentiation of satellite cells, which are essential for muscle repair and development, driving both muscle hyperplasia and hypertrophy