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In Vitro Semax (25-100 M, 48 h) can reduce the levels of A oligomers [1]

5HT receptor activation alleviates neuropathic painrelated antidepressant via the ACcAMPPKAMEKCREBBDNF cascade [26], and oxytocin protects dopaminergic function against corticosterone through PKA/CREB signaling [27]

Dihexa: Goes beyond modulation to build new synapses

From a therapeutic perspective, anti-inflammatory drugs may partially restore neurogenesis and represent an avenue for adjunct therapy in ameliorating neuronal decline [110]