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sFASL binds to the Fas receptor (CD95) on the surface of target cells, activating the caspase cascade and triggering programmed cell death

The lipid peroxidation end product, 4-hydroxynonenal (4-HNE), also increased in infected lungs, peaking on day seven (Fig

As we observe that STIM2, Orai1, and Orai3 proteins expressions are reduced by this treatment, reduced coupling between STIM2 and Orai more likely explains the reduced Ca 2+ entry after P 2 Y receptors activation than an inhibition of Ca 2+ release from the ER

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