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To ascertain whether the induced cellular death resulting from this treatment was attributable to ferroptosis, we added various cell death inhibitors, including ferrostatin-1 (Fer1), a specific ferroptosis inhibitor, necrostatin-1 (Nec1), a necrosis inhibitor, and Z-VAD-fmk (Z-VAD), an apoptosis inhibitor, to the cancer cells treated with IFN and etomoxir

Once transported into the microbe, the inhibitors then block the production of TMAO by inactivating a specific gut microbe enzyme called cutC (choline utilization protein C)

Brain function is especially vulnerable when mitochondrial energy becomes inconsistent

Transports fatty acids