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Description

Reactive oxygen species (ROS) may directly alter vascular function or cause changes in vascular tone by several mechanisms including altered nitric oxide (NO) bioavailability or signaling

Inhibition of PERK-mediated unfolded protein response acts as a switch for reversal of residual senescence and as senolytic therapy in glioblastoma

It may also support the gut-brain axis by influencing neurochemical pathways and promoting communication between the gastrointestinal tract and central nervous system (CNS)