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With RSL3-induced reduction in pNPM1 levels, DNA damage sites (marked by -H2AX foci) were not repaired due to limited availability of pNPM1

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Therefore, targeted inhibition therapy of REST-mediated ferroptosis may serve as a novel therapeutic strategy for ameliorating AKI and its transition to CKD

Potential adverse or off-target effects associated with chronic Src kinase inhibition warrant consideration, given broad regulatory role of Src family kinases in diverse cellular processes such as proliferation, differentiation, immune function, and metabolism [1]