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Diazinon group hepatocytes exhibited severe vacuolar degeneration in the centrilobular and peri-portal regions (fig

Furthermore, the cardiolipin in the outer mitochondrial membrane pulled -syn monomers from oligomeric fibrils and enabled their refolding back into -helices, thereby buffering their aggregation and resultant pathological impacts (Ryan et al., 2018)

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Contrastingly, the cumulative release of Fe 3+ at pH 6.0 reached 40.2% after 24 h and further significantly increased to 60% with the addition of HAase, which may be attributed to the accelerated pH-induced degradation of the Fe-TA MPNs following HAase-mediated HA disassembly, thus facilitating the diffusion of Fe 3+