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These molecules are found in abundance in eukaryotic cells and have been identified as playing a critical role in tumour metabolism in various cancers

To inhibit GPX4, we used RSL3 and ML210 that covalently bind the selenocysteine residue (Sec46) of GPX4 leading to its inactivation and degradation 31

The viability of WT HCT116 and Nalm6 cells treated with FA was significantly reduced in the presence of L-BSO, indicating that GSH synthesis contributes to cellular FA tolerance (Fig

Given the absence of long-term human trial data, conservative use under clinical supervision is the standard position among practitioners who take the evidence seriously