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BPC-157 significantly accelerated tendon fibroblast outgrowth, increased cell survival under oxidative stress, and enhanced cell migration in a dose-dependent manner, effects attributed to activation of the FAK-paxillin pathway central to tendon repair

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In the four-phase concept, there are the hemostasis phase, the inflammatory phase, the proliferation phase, and the remodeling phase

This pathway activation explains why BPC-157 shows particularly strong results in tendon and ligament repair models across the BPC-157 vs TB-500 comparison literature