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Zhou F, Yu T, Du R, Fan G, Liu Y, Liu Z, et al

Increased levels of cytoplasmic ROS are generated through increased metabolic activity, mitochondrial dysfunction and oncogene activity which drive critical signalling pathways to sustain hyperproliferative growth 4 and inflammation 3

the simultaneous presence of mitochondrial abnormalities in cardiac and neural tissues of comorbid patients, suggesting a potential shared cellular defect