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All compounds at peak coordinated activity

Taken together, these data showed that addition of N-glycan on N92 site of Ea-domain prevented the degradation of proIGF-1Ea by the proteasome, probably overcoming the folding limitation of Ea-domain and the tendency of unglycosylated proIGF-1Ea to self-aggregate

These actions collectively orchestrate a more efficient and organized repair process in preclinical models