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To cope with this, these cells are equipped with several defense mechanisms which include different DNA damage repair pathways (Fig

Treatment with marine collagen peptides modulates glucose and lipid metabolism in Chinese patients with type 2 diabetes mellitus

HIV-1 Tat-mediated microglial senescence by miRNA-505-SIRT3 pathway Senescence was initially defined as the cessation of cell division after multiple replication cycles [62] and has expanded to include premature senescence triggered by telomere shortening, oxidative stress, mitochondrial dysfunction, and lysosomal alterations [63]