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They revealed that only the benzamide compound BML-210 produced a significant increase of FXN mRNA expression (Herman et al., 2006), although other HDAC inhibitors showed a more pronounced increase of histone acetylation without any increase in FXN expression, indicating a degree of compound specificity for FXN gene silencing (Table 2)

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The key to this architecture is that the Purkinje cell stripes and their associated climbing fiber inputs operate as synchronous units [361, 362]

Multiple mechanisms have been suggested for ES in the improvement of nerve regeneration