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In this article, we first review the mechanisms underlying the abnormal intrinsic immune activation and programmed cell death in GN and then describe the characteristics and methods used to develop animal and cell models of GN caused by elevated uric acid and deposited sodium urate crystals

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However, indirect evidence from meta-analyses suggests that NAC demonstrates more consistent benefits across various liver conditions, likely due to its superior bioavailability and ability to effectively raise intracellular glutathione levels