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By employing tumour -specific targeting, controlled release mechanisms, and multifunctional designs, these systems offer a pathway to balance the dual roles of oxidative stress in cancer progression and treatment

Potential of these effects may be heightened in patients with breast cancer treated with hepatically metabolized chemotherapeutics (e.g., cyclophosphamide, paclitaxel, anthracyclines) or tyrosine kinase inhibitors in which drug-drug interaction-based elevation or lowering of enzyme activity can result in supra-therapeutic drug concentrations, accumulation of toxic metabolites and compromised detoxification of reactive oxygen species facilitating hepatocellular distress

Circadian and light-induced transcription of clock gene Per1 depends on histone acetylation and deacetylation

Management of Complicated Skin and Soft Tissue Infections with a special focus on the role of newer antibiotics