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This finding agrees with the literature whereby the role of quinone redox metabolism is an ambiguous source of toxicity, with evidence suggesting that redox cycling requires supra-pharmacological concentrations of doxorubicin to generate substantial ROS in tissues and cells 10

The results mentioned above, along with the strong association between GPX7 and several immunosuppressive cells (e.g., NKT cells, TAMs, and MDSCs) or inhibitory immune checkpoint markers (e.g., PD-1, CTLA-4, LAG-3, TIGIT, TIM-3 and CD96 ) further enhances the importance of GPX7 in the regulation of an immunosuppressive tumor microenvironment 158,159,160,161,162

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encoding ferroportin) that prevent hepcidinferroportin binding also cause haemochromatosis