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Description

Research has confirmed that MET inhibitors such as capmatinib and tivantinib can maintain the stability of PD-L1 expression by inhibiting GSK3-driven phosphorylation and the subsequent breakdown of PD-L1

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The lyophilized state prevents hydrolytic degradation and maintains peptide bond integrity

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Transected rat Achilles tendons treated with the peptide showed not only faster healing but qualitatively better tissue organization

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Different mechanisms, complementary targets

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