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1,7-9 There is general agreement, however, that abnormal processing of pain signals in the brain (central sensitization) and heightened sensitivity along peripheral pain pathways are key mechanisms underlying fibromyalgia

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These molecules have a strong affinity for the estrogen receptors (ERs) ER, ER, and G-protein coupled ER1 (GPER1), through which, after binding, induce a direct or indirect genomic signaling to change the expression pattern of several genes (Fuentes and Silveyra, 2019)

Our goal is to find the most effective way to slow cell turnover with the fewest side effects possible