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Moreover, zinc suppresses Th17 cells by inhibiting glutaminolysis, which serves as the primary energy source for these cells, thereby lowering the risk of autoimmune diseases, such as autoimmune encephalomyelitis [333]

We subsequently analyzed the action of the PERK inhibitor GSK and the HSP90 inhibitors luminespib and 17-AAG to show that certain compounds other than PDD also act as enhancers of targeted protein degradation, thereby demonstrating the usability of our proposed strategy

This approach could reveal novel regulatory mechanisms and potential therapeutic targets within the complex interplay of lipid metabolism and cell death pathways

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