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An open-label, dose escalation, multi-center phase 1 study of PRLX 93936, an agent synthetically active against the activated ras pathway, in the treatment of relapsed or relapsed and refractory multiple myeloma

Antioxidant therapy inhibits the TRPV1 channel by overexpressing GSH and GSHPx, while downregulating GSH and GSHPx promotes the TRPV1 channel [50,51,52]