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Given this functional redundancy, the inability of the WT H1.4 allele or other H1 isoforms to compensate for the RS mutation has been interpreted as evidence for a dominant-negative or gain-of-function effect 2

T Serikawa, O Fujii, and other staff members of Institute of Laboratory Animals, Kyoto University, Graduate School of Medicine, for careful animal maintenance, M Sugimoto for secretarial assistance in the preparation of this manuscript, C Sayo, A Yamaguchi, T Matsushita, and I Fujiwara for their technical assistance, Cell Resource Center for Biomedical Research, Institute of Development, Aging and Cancer, Tohoku University and Dr

3 Sun X, Liu C, Shi Y, Li C, Sun L, Hou L, Wang X

together they amplify the GH pulse more than either arm alone