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Furthermore, nanomedicine presents opportunities for neuroprotective strategies by employing antioxidant and anti-inflammatory NPs to protect and repair damaged neurons

This section delineates SFNs multifaceted mechanisms across epigenetic regulation, programmed cell death pathways (particularly ferroptosis), and tumor immune microenvironment remodeling, establishing its role as a truly multi-targeted therapeutic agent

A study proved that p53 could bind to dipeptidyl-peptidase 4 (DPP4) to reduce the DPP4-dependent oxidization of PUFAs to inhibit ferroptosis in colorectal cancer cells[46]