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The RAS has been associated with neuropsychiatric and neurodegenerative disorders, including Parkinsons disease (PD), stroke, and Alzheimers disease (AD)

Initiation of neuroinflammation occurs with microglial activation using toll-like receptors (TLRs) and NLRP3 inflammasomes, resulting in elevated interleukin release (IL-1, TNF-, and IL-6) affecting the vasculature and the bloodbrain barrier, leading to vasogenic edema [89]