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164 It was proved that Temporin-1CEa could co-localize with P-gP on the surface of drug-resistant cell membranes, so the anticancer mechanism of Temporin-1CEa may be acting on P-gP and affecting its efflux function, thereby reversing multidrug resistance in tumor cells

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Although effective with supervised dosing, it is no longer first-line therapy due to poor adherence, variable efficacy, and hepatotoxicity concerns (168, 296)