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TNF could promote the proliferation of synoviocytes and lead to the progression of synovitis, and the role in reducing ferroptosis sensitivity to lessen the death of FLSs may also be a manifestation of this mechanism

Moreover, we found that expression of both CLV3 and WUS genes were maintained as correctly positioned separate domains, further supporting the idea that these domains are established using positional cues rather than a dependency on cell lineage

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Crossbreeding Gprc5a -ko mice with SPC-dnIB- mice to generated Gprc5a / /SPC-dnIB- mice, in which dominant negative (dn) or super-repressor (SR) IB gene is driven by the type II cell marker surfactant protein C (SPC) promoter [30]