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OsNCED5, a 9-cis-epoxycarotenoid dioxygenase gene, regulates salt and water stress tolerance and leaf senescence in rice

Together, these findings suggest that ferroptosis contributes to HD pathology, and its inhibition may represent a viable strategy for disease modification ( Figure 9 )

This decay in protein function in response to DFO treatment or NFS1 or ISCU knockdown corresponded with a gradual loss of mitochondrial Fe-S protein expression (Fig

We previously identified a cell-size mutant, suppressor of mat3 15-1 ( smt15-1 ), that has elevated cellular glutathione, increased number of cell divisions, and small daughter cells