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Mol Brain 4:113 Zhang Y, Wang M, Chang W (2022) Iron dyshomeostasis and ferroptosis in Alzheimers disease: molecular mechanisms of cell death and novel therapeutic drugs and targets for AD

In the process of practical application, researchers should understand the species differences between experimental animals and human beings, the formation mechanism of MASLD model and pathological changes are very different from those of human beings when they suffer from the disease and they need to select appropriate models according to specific research purposes and needs and consider the complications of the MASLD model

Beyond repression of Nrf2: An update on Keap1

The system that removes these toxic reactive oxygen species includes a host of enzymes: Glutathione peroxidase (GPX): GPX detoxifies peroxides with glutathione acting as an electron donor in the reduction reaction, producing glutathione disulfide as an end product