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Inflammopharmacology 112

[DOI] [PMC free article] [PubMed] [Google Scholar] 108.elik N, Vurmaz A, Kahraman A

Perng BC, Chen M, Perng JC, Jambazian P

Hypertensive disease is in most patients due to increased sympathetic nerve activity ( 1 AR-selective antagonists may be explained by their ability to hamper norepinephrine release through presynaptic modulation, in agreement with previous analyses showing that the hypotensive effect of AR antagonists, including atenolol, did not depend on cardio depression or suppression of renin secretion ( 1 AR-selective antagonists in such cardiac conditions may therefore include also their ability to lower norepinephrine release