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Linking alpha-synuclein phosphorylation to reactive oxygen species formation and mitochondrial dysfunction in SH-SY5Y cells

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Pharmacological suppression and genetic ablation of these channels ameliorate the inflammatory and degenerative retinal phenotypes associated with ocular hypertension (119), suggesting possible roles in glaucoma

The ROS produced upon engagement of these receptors can then alter downstream signaling by oxidizing specific cysteine residues within a variety of intracellular proteins including protein kinases, protein phosphatases, adaptor proteins, calcium channels, and transcription factors