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Description

Longitudinal study of oxidative status in 312 cystic fibrosis patients in stable state and during bronchial exacerbation

In several cancer cells, the expression, and the activity of ERR, and its cofactor PGC-1, is further influenced by oncogenic signals (e.g., IGF1 receptor pathway, estrogen signaling, mTOR pathway) and induces metabolic programs favoring cell growth and tumor progression

Redox-crippled MitoQ potently inhibits breast cancer and glioma cell proliferation: A negative control for verifying the antioxidant mechanism of MitoQ in cancer and other oxidative pathologies