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The cardiac lymphatic system stimulates resolution of inflammation following myocardial infarction

Using an AKT activator (SC-79) and an AMPK inhibitor (HY-151361) to simulate the over activation of tumor pathways, we observed a significant increase in the precursor and active forms of SREBP2 (Fig

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They introduced ferroptosis as an iron-dependent, non-apoptotic form of regulated cell death [14]