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In summary, these findings suggest that inhibiting ROS with NAC downregulates HIF-1 and BNIP3/NIX-mediated mitophagy, thereby exacerbating cell apoptosis and inflammation in M9a+sh EPAS1 cells

Some studies suggest that CNTF may indirectly contribute to microglial activation, especially during chronic inflammation or injury, but it is not a classical microglial activator, such as IL-1, TNF-, or LPS [106]