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Dnmt1, Dnmt3a and Dnmt3b) and the demethylases teneleven translocation (TET) methylcytosine dioxygenase (Tet13), and the methyl-CpG-binding protein 2 (MeCP2)

Missense mutations in GPCRs can mislead protein folding and post-translational modifications to cause trafficking alterations, in which pharmacological chaperones are applicative therapeutic regimens

Its association with various metabolic irregularities, such as the onset of atherosclerosis, high lipid concentration, obesity, and liver lipid accumulation, is widely recognized (81)