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This further exacerbates ROS production and the ensuing oxidative stress [17], while also leaving the cells of the RPE with insufficient energy to divide and carry out their normal functions

This two-step testing ensures that any subsequent clinical decisions are based on robust evidence, reducing the risk of misdiagnosis

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Furthermore, an increased plasma von Willebrand factor is found in hyperhomocysteinemic subjects bearing premature arterial disease (Freyburger et al, 1997) and, of major relevance, acute methionine loading induced moderate hyperhomocysteinemia results in a significant increase in von Willebrand factor (Constant et al, 1999)