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During PD progression, compromised intestinal barrier integrity with increased permeabilitytermed leaky gut 71 enables translocation of pro-inflammatory microbial products such as lipopolysaccharide (LPS) and cytokines into the bloodstream, thereby triggering systemic inflammation 72,73

Immediately after the discovery of PCSK9 protein and its function, growing evidence from genetic association studies showed PCSK9 inhibition as a potential lipid-lowering target [15]

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FTO level is upregulated in PD model, while FTO silencing alleviates cell ferroptosis