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Administering 75 mg/kg/day of FA to high-fat-fed LDL receptor-deficient (LDLR/) mice for 16 weeks resulted in a significant inhibition in atherosclerosis plaque progression and vascular smooth muscle cell (VSMC) dedifferentiation by modulating the mTOR/p70S6K signaling pathway, which involved the reduction in lipid levels and the suppression of oxidative stress and inflammatory response [82]

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