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Although the specific pathogenesis of ADHD has not yet been elucidated, neurotransmitter abnormalities (dopamine (DA), norepinephrine (NE), 5-hydroxytryptophan (5-HT)) as well as increased OS and neuroinflammation in patients with ADHD have been the focus of most research (Curatolo et al., 2009)

Despite immense therapeutic potential, the unfavorable pharmacokinetic properties of flavonoids limit their use in clinical settings (Hussain et al., 2025)

Furthermore, HDL drives conversion to the anti-inflammatory M2 macrophages which have enhanced efferocytosis ability compared to inflammatory M1 macrophages (56,208) leading to increased plaque stability

Diabetes Care 31 , 1679 (2008)