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The picture that emerges from these experiments is that endogenous GSH is not just an inhibitor of inflammation, but it is required to allow a proper response to infection, and direct the migration of inflammatory PMN away from the lung, where they cause ARDS, and towards the site of infection, where they kill bacteria (Figure 6)

If you are a current patient at RCBM, please discuss with your clinician about treatment options at RCBM and how you can include trauma therapy in your treatment plan