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Congenital hemolytic anemias attributed to defects in the biosynthesis of GSH (in the genes of GCL, GCS and glutathione reductase) highlight the key role of GSH in RBCs homeostasis [197,198]

Meanwhile, another NOS3 polymorphismthe 27-bp VNTR (4b/a)showed a trend toward association in younger patients (50 years) without COPD, with the 4b allele and 4b/4b genotype more frequent in severe cases (p values around 0.060.07), though these findings did not reach conventional statistical significance

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Its reduced (NADH) and phosphorylated forms (NADP+ and NADPH) are as important as NAD+ (1-2)