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The finding that GSH can enter the ER by facilitated diffusion through the Sec61 translocon in a yeast mutant [36] is congruent with this model, even though it should be acknowledged that the toxic ~ 10-fold increase in cytosolic GSH in this yeast mutant represents a rather non-physiological situation with regard to glutathione gradients at intracellular membranes

doi: 10.1210/jcem.86.4.7414

An Integrated Study on the Antitumor Effect and Mechanism of Triphala against Gynecological Cancers Based on Network Pharmacological Prediction and In Vitro Experimental Validation

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