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[3] This is a known effect and not cause for concern

However, under conditions of sustained tumoral stress and prolonged receptor activation, we propose that nuclear accumulation of ATF4 may render it more susceptible to turnover mechanisms such as proteasomal degradation, which is in contrast to its canonical role initiating integrated stress response (ISR) [58, 59]

Prez-Fuentes N, Alvario R, Alfonso A, Gonzlez-Jartn J, Gegunde S, Vieytes MR, et al.Enniatins A1 and B1 alter calcium homeostasis of neuronal cells leading to apoptotic death