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Primarily, its poor aqueous solubility, chemical instability, and narrow therapeutic window where supraphysiological levels may induce neurotoxicity (Mancuso, 2017)

Juglanin has been reported to suppress the PI3K/AKT signaling pathway, resulting in a reduction of anti-apoptotic proteins (Bcl-2 and Bcl-xL) and an increase in pro-apoptotic proteins (Bax and Bad)