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CAEBV was previously believed to induce lymphocyte cytotoxicity, such as HLH, through systemic inflammation and clonal proliferation of EBV-infected T or NK cells

Apolipoprotein C-III and the metabolic basis for hypertriglyceridemia and the dense low-density lipoprotein phenotype

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Collectively, these findings underscore a close association between the hypoxic microenvironment and the initiation, progression, and severity of SLE