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Studies (albeit predominantly in mice) have pointed to a number of factors including increased oxidative stress in the hair follicle 9 , depletion of melanoblast stem cells from the niche 10 , apoptosis of differentiated matrix melanocyte in the hair bulb 11 and DNA damage repair (DDR) abnormalities, that all may contribute in part to hair greying 12

Reducing ferroptosis might suppress inflammation and be beneficial to the generation of pro-angiogenic factors, thereby improving healing of wounds

KPV exerts anti-inflammatory effects through mechanisms involving melanocortin receptor signaling and modulation of inflammatory mediator production

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