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Oxid Med

Both NO and CO activate soluble-guanilyl cyclase (sGC), thus inducing and increase in intracellular cyclic-guanilyl monophosphate (cGMP) as second messenger [8, 9]

Additional data from Japan and the U.S

Conversely, other studies propose that while excessive HO-1 activation might protect against heme-induced oxidative damage, it may also trigger atypical ferroptosis by generating excessive free iron through heme degradation ( Figure 4 )