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Here several hypotheses are suggested, including persistent infections, reactivation of endogenous microbial reservoirs, infection-triggered autoimmunity, or other persisting post-infectious immune dysfunctions (Komaroff and Bateman, 2021

Acknowledgements We are grateful to CAPES, FAPESP and GlaxoSmithKline for their financial support

This strategically shifts the detection window away from the highly positive regions where most common endogenous species oxidize, thereby securing the high selectivity required for reliable clinical diagnostics [170]

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